However, it’s important to understand that Botox is not a permanent fix. It suggests that a moderate dose of Botox helps reduce spasticity long enough to allow for functional improvements without causing a significant decrease in strength.
One study found that reduction in spasticity was associated with significant improvement in arm function after stroke. This prevents the muscle from spasming and will allow it to stay more relaxed. Botox (botulinum toxin A) works as a “nerve block” that blocks the release of chemicals that signal your muscles to tighten. This can affect range of motion and limit performance of many daily activities.īy injecting Botox directly into the muscles that are affected, spasticity can be significantly reduced. Contracture occurs when a muscle is unable to fully relax and elongate, resulting in the muscle becoming tighter and shorter over time. Over time, spasticity can be painful and can lead to other complications such as contracture. This miscommunication can cause multiple muscles to contract involuntarily without being able to relax, causing spasticity. When a stroke damages the motor cortex, it impairs the brain’s ability to tell the muscles to either contract or relax. Patients may feel like spasticity is a problem with the muscles themselves, but the problem actually stems from miscommunication between the brain and the muscles. Spasticity is a condition where the muscles become stiff due to prolonged contraction or spasm after neurological injuries like stroke. This article will discuss the use of Botox for spasticity and help you understand how to turn your temporary gains from Botox into lasting improvement. Some patients aren’t provided with enough education on exactly how Botox works, so they only see short-term results. One way to treat spasticity and relax the muscles is to use Botox for stroke patients. Following a stroke, miscommunication between the brain and muscles can cause the muscles to involuntarily contract and become stiff, a condition known as spasticity.
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